Abstract
Acknowledgments
Notes
References
Table 1
(ERS: endoplasmic reticulum stress, TG: thapsigargin, UPR: unfolded protein response, GRP: glucoseregulated protein, C/EBP: CCAAT/enhancerbinding protein, eIF: eukaryotic initiation factor, JNK: c-Jun N-terminal kinase, IRE1: inositol-requiring enzyme type 1, PERK: protein kinase R [PKR]-like endoplasmic reticulum kinase, XBP: X-box binding protein, LC3: microtubule-associated protein 1A/1B-light chain 3, 4PBA: 4-phenylbutric acid, ATF: activating transcription factor, SEPS: selenoprotein S, CHOP: C/EBP homologous protein, HSP: heat shock protein, TRAP: triiodothyronine receptor auxiliary protein, PDL: periodontal ligament, ER: endoplasmic reticulum, RANKL: receptor activator of NF- κB ligand, miR: microRNA, siRNA: small interfering RNA, PEI-NPs: polyethylenimine nanoparticles, VEGF: vascular endothelial growth factor, CTGF: connective tissue growth factor, Bcl-2: B-cell lymphoma 2, XBPs: X-box binding protein spliced, ACC: adenoid cystic carcinoma, OSCC: oral squamous cell carcinoma, STAT: signal transducer and activator of transcription, LIF-R: leukemia inhibitory factor receptor, IL: interleukin, IL [number] R: interleukin [number] receptor, NF-κB: nuclear factor-kappa B, SREBP1: sterol regulatory element-binding protein 1, CREB3L3: cyclic AMP responsive element-binding protein 3-like 3, DDIT: DNA damage inducible transcript, HTRA: high-temperature requirement A, DSPP: dentin sialophosphoprotein, SERCA: sarco/endoplasmic reticulum Ca2+-ATPase, MMP: matrix metalloproteinase, PCNA: proliferating cell nuclear antigen)