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Kim, Park, and Seok: A Case Report of Thyrotoxic Periodic Paralysis; Serial Nerve Conduction Studies before and after Recovery
Original Article

Korean J Community Nutr 2015;17(2):98-100.

Published online: 18 December 2015

DOI: https://doi.org/10.14253/KJCN.2015.17.2.98

A Case Report of Thyrotoxic Periodic Paralysis; Serial Nerve Conduction Studies before and after Recovery

Min Suck Kim, Jung A Park, Jung Im Seok

Department of Neurology, Catholic University of Daegu, School of Medicine, Daegu, Korea

Address for correspondence; Jung Im Seok Department of Neurology, School of Medicine, Catholic University of Daegu, 33 Duryugongwon-ro 17-gil, Nam-gu, Daegu 42427, Korea Tel: 82-53-650-3440 Fax: 82-53-654-9786 E-mail: ji-helpgod@hanmail.net

Received 20 May 2015       Revised 12 August 2015       Accepted 20 August 2015

Copyright © 2015 Journal of the Korean Society of Community Nutrition

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

Keywords: Hypokalemic periodic paralysis, Thyrotoxicosis, Electrodiagnosis

REFERENCES

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3.Rajshekher G., Kumar S., Prabhakar S. Reversible electrophysiological abnormalities in hypokalemic periodic paralysis. Indian Pediatr. 2008. 45:54–55.
4.Tran HA. Thyrotoxic periodic paralysis. Mayo Clin Proc. 2005. 80:960–961.
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5.Cheng CJ., Kuo E., Huang CL. Extracellular potassium homeostasis: insights from hypokalemic periodic paralysis. Semin Nephrol. 2013. 33:237–247.
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6.Kuwabara S., Kanai K., Sung JY., Ogawara K., Hattori T., Burke D, et al. Axonal hyperpolarization associated with acute hypokalemia: multiple excitability measurements as indicators of the membrane potential of human axons. Muscle Nerve. 2002. 26:283–287.
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7.Cheng CJ., Lin SH., Lo YF., Yang SS., Hsu YJ., Cannon SC, et al. Identification and functional characterization of Kir2.6 mutations associated with non-familial hypokalemic periodic paralysis. J Biol Chem. 2011. 286:27425–27435.
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Table 1.
Motor nerve conduction study before and after correction of serum potassium level
During paralysis After recovery
Nerve CMAP amplitudea (mV) Conduction Velocityb (m/s) Distal latency (ms) F wave Latency (ms) CMAP amplitudea (mV) Conduction Velocityb (m/s) Distal latency (ms) F wave Latency (ms)
R L R L R L R L R L R L R L R L
Median 7.9 4.9 65.8 63.6 2.6 2.9 24.9 24.8 14.1 13.0 70.1 64.5 2.8 2.9 23.9 23.4
Ulnar 6.2 5.9 70.8 65.4 2.3 2.2 26.2 24.9 14.9 12.4 72.1 65.4 2.1 2.1 25.1 25.1
Tibial 0.1 3.3 46.4 46.4 5.0 4.9 NR 55.9 10.4 10.2 50.0 52.1 3.8 3.8 48.9 49.4
Peroneal 0.5 5.8 53.0 54.4 5.1 5.8 NR 52.6 6.7 5.4 56.9 54.7 4.5 4.6 44.6 46.6

Abnormal values are in bold.

a CMAP amplitude was measured from the positive peak to the negative peak (peak-to-peak) and obtained by distal stimulation

b Median and ulnar nerve conduction velocity was measured in the elbow to wrist segment and tibial and peroneal nerve conduction velocity was measured in the knee to ankle segment. CMAP; compound motor action potential, R; right, L; left, NR; no response.

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