Sialadenosis in a Patient with Alcoholic Fatty Liver Developing after Heavy Alcohol Drinking

Yeon Hwa Yu; Young Sook Park; Seong Hwan Kim; Byoung Kwan Son; Dae Won Jun; Yun Ju Jo; Yong Suk Ryu; Hyeon Suk Kim

doi:10.4166/kjg.2009.54.1.50

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Yu, Park, Kim, Son, Jun, Jo, Ryu, and Kim: Sialadenosis in a Patient with Alcoholic Fatty Liver Developing after Heavy Alcohol Drinking

Original Article

Korean J Gastroenterol 2009;54(1):50-54.

Published online: 21 February 2009

DOI: https://doi.org/10.4166/kjg.2009.54.1.50

Sialadenosis in a Patient with Alcoholic Fatty Liver Developing after Heavy Alcohol Drinking

Yeon Hwa Yu, M.D., Young Sook Park, M.D., Seong Hwan Kim, M.D., Byoung Kwan Son, M.D., Dae Won Jun, M.D., Yun Ju Jo, M.D., Yong Suk Ryu, M.D., Hyeon Suk Kim, M.D.^∗

Department of Internal Medicine, Eulji University School of Medicine, Seoul, Korea

^∗Department of Radiology, Eulji University School of Medicine, Seoul, Korea

Correspondence to: Young Sook Park, M.D. Department of Internal Medicine, Eulji University Eulji General Hospital, 14, Hangeulbiseok-gil, Nowon-gu, Seoul 139-711, Korea Tel: +82-2-970-8207, Fax: +82-2-970-8621 E-mail: pys1109@eulji.ac.kr

Received 28 January 2009 Accepted 31 January 2009

Abstract

Sialadenosis is a unique form of non-inflammatory, non-neoplastic bilateral salivary gland disorder characterized by recurrent painless swelling which usually occurs in parotid glands. Alcoholism is one of the main causes of sialadenosis along with diabetes, bulimia, and other idiopathic causes. The prognosis is verified according to the degree of liver function. We present a case of a 46 year-old man who had alcoholic fatty liver disease diagnosed as alcoholic sialadenosis based on clinical points of recurrent bilateral parotid swelling after heavy alcohol drinking, computed tomography, and fine-needle aspiration biopsy. After stopping alcohol drinking and treated with conservative treatment, he got improved without specific sequela.

Keywords: Fatty liver, Alcoholic, Parotid gland, Sialadenitis

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Fig. 1.

Abdominal CT scan showed no abnormal pancreas.

Fig. 2.

Positron emission tomography-computed tomography (PET-CT) findings. (A) PET-CT finding showed diffuse hypermetabolism in bilateral parotid gland. (B) Yellowish arrows indicated hypermetabolism in bilateral parotid gland.

Fig. 3.

CT scan showed enlarged bilateral parotid gland with ho-mogeneous density.

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